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puma vga nsd plau oas

 
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 PostWysłany: Pią 7:29, 11 Mar 2011    Temat postu: puma vga nsd plau oas Back to top

Comprehensive transient amnesia


The exact mechanism on the TGA pathogenesis is unknown so far, has not been a TGA of the pathology report, but its damage is known mainly for the anatomy of the limbic system. The hippocampus, paraneoplastic mammillary bodies, which are related to the structure and recent memory. The group of 6 patients with history of hypertension,[link widoczny dla zalogowanych], 3 patients had a history of coronary heart disease. Fundus arteriosclerosis 5 cases, 3 down TCD examination showed a vertebral basilar insufficiency. Most (9 / 11) over the age of 50, rejection of vasodilator therapy better soon. It can cause a brain artery that had ischemia induced a large study suggests that TGA Department of posterior cerebral artery branch or vertebral basilar artery system of a TIA, vascular disorders support this theory. By CT scan was feeding the bulk of the TGA patients with TIA and without neurological disease (NND) study suggests that TGA patients were compared because of ischemic, can be seen as a less hazardous TIA. TGA is ~ had sex. Some may relapse, it was suggested that mechanisms of epilepsy immunogenicity carbuncle], but most of the current study does not support this view. The group has done a EEG5 7 Ba Ba normal crossing t slow increase in the other 2 cases no carbuncle discharge. 1 2 recurrence were feeding no abnormal EEG investigation, all eight patients were not treated to a little anti-epileptic, it can be other than epilepsy, does not support the theory of lame immunogenicity. Forehead wins lame attack leaves is the onset of the patient's forgotten a short time, and with automatic storage disorder, personality disintegration,[link widoczny dla zalogowanych], TGA patients without these characteristics. Foreign scholars [5 collected 117 cases of 120 people in the TGA and the onset of the late onset of EEG,[link widoczny dla zalogowanych], the results show that the TGA attack EEG is usually normal, no carbuncle discharge,[link widoczny dla zalogowanych], indicating that the TGA does not exist to win epilepsy mechanism. In this paper, 1 case of 42-year-old female patient, a history of migraine for 13 years, working in the sudden headache, 10rain after the sudden loss of memory, a diagnosis of TGA, such as the treatment given pizotifen no recurrence. Another 65-year-old female patient in 1 case. The right temporal pain and oblivion simultaneous fundus arteriosclerosis. EEG showed coronary heart disease, no recurrence. TGA and the relationship between migraine? Small number of our cases is difficult to explain the recent emphasis on history of migraine abroad. TGA found in adult patients 25 to 30 with migraine headaches can occur even in the attack, research suggests that vascular spasm may be the basis for the cause of TGA, but not sure the relationship between migraine and TGA 2.5TGA Treatment and Prognosis 1O l1 cases in this group patients according to T / a, treatment, and 1 by migraine, 8-resistant ranging from 1 to 3 years follow-up, only 1 patient had recurrence 2, but also other areas does not appear abnormal. TGA generally good prognosis. Recurrence rate is low, is superior to TIA. Recurrence rate has been reported in the 20 or so, our data further. Difficult to draw conclusions on the prognosis. TGA may be due to the many causes, so the prognosis depends on the underlying disease to determine, for old age, hypertension, hyperlipidemia patients, prevention should focus on underlying diseases such as cardiovascular diseases. TGA should be any detailed examination of the patient, so as to make a more accurate prognosis of the valuation. Integrated above. We believe that TGA may be a result of the many causes of a clinical syndrome, not a separate disease; forgotten attack the main basis for the diagnosis of TGA; all suspected medical consultation should be done per head CT, EEG,[link widoczny dla zalogowanych], TCD checks to Identification of epilepsy and other diseases, and win; most patients the cause of the vascular immunogenicity, and can be seen as a less hazardous TIA


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