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Compound epicatechin inhibited the tumor promotion by phenobarbital
hemistryofGGTinratiivetduringAFB. inducedcarcinogenesisJNatlCantefInst197555579 one - one by one 8. Tan Xiaodi. And so on. Micro-computer-aided liver disease delete volume and stereological estimates of Biomedical Engineering 1989; 8 70.9 Zhang Yingchun,mbt scarpe prezzi, et al. Diethylnitrosamine-induced liver cell staining variation of phenotype. Journal of Pathology l989; I823OPitotHCPrinci01esofcancerbiology: chemicalcarcinogenesisInDevitaJr. VetaI_Cancer. principlesandpratticeofoncologY, 2ndEd. VolIPhiladetohia: JBLippincottCompany, 1985:79 lRuchRJ,clarks shoes outlet, etaL. PreventionofCytOtOXicityandinhibi-tionofintercellularcommunicationbyantioxidamcatechinskolatedfromchinesegreentea. Carcinogenesisl989; 10:1003 (1990 年 4 月 24 日 Received I4 August the same year, at Revised InhibitioneffectofepicatechinmlphenolmrbitolinducedproliferationofprocancerousHvercellsDingLianetalDepartmemofPathology,asics shoes online, FacultyofBasicMedicine.PekingUnionMedicalCollegeandInstituteofBasicMedicalSciences,scarpe nike 2011, ChineseAcademyofMedicalsciences, Bejing1oo73oEpicatechincomplexwasgiventodiethylnitrosamine-initiatedmaleWistarratsconcomitantlywiththeadministrationofphenobarbitolduringhepatocarcinogenesis.Theepicatechincomplexwasex.tractedfromLongjinggreenleaandhsconcentrationofwholeepicatechinswas87.5%. Theresultsdenotedthatepicatechinefficientlyinhibitedthepfomotiveeffectofphenobarbito1.HighperformanceIiauidchromographyanalysisproovedthatthechemicalpropertiesofphenobarbitolandepicatechinwel-estillwellpreservedwhenthesetwosubstancesweredissolvedtogetherindistilledwater.Itwasnotedthattherewasriodirectchemicalreactionbetweenthesetwosubstances.Theinhibitionmechanismandneedoffurth stare studiesconcerningtheeriectofepicatechinoncellmetabolismarediscussed.keywordscatechinphenobarbitallivermorphometry
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